• MySport is a genetic test designed to evaluate the genetic profile linked to sports performance, helping to achieve fitness objectives and sports goals.

• This analysis examines genetic markers supported by scientific evidence, providing personalized recommendations to optimize feeding and training plans.

• Knowing the genetic profile of an athlete allows you to identify their strengths and limitations, which facilitates the design of a personalized training plan. This includes factors such as level of physical activity, exercise intensity, fitness goals and predisposition to injury, which optimizes results and minimizes risks.

Genetic sports performance profile

Athletic potential

Resistance
It evaluates aerobic capacity, fatigue resistance and maximum oxygen consumption (VO2max). Key genes: ACE, ACTN3, ADRB2, BDKRB2, CKM, COL5A1, CRP, HIF1A, IL6, NRF2, PPARa, VEGFa.

Strength
Determines the ability to generate explosive force and muscle development.Key genes:

ACTN3, ACE, ADRB2, BDKRB2, GDF5, MTHFR, PPARa, PPARg, VEGFa.

High intensity and sprints:

The favorable genotypes in ACTN3, ADRB2, and NOS3 Will improve explosive potency and cardiovascular response in short efforts.

Predisposition : Injury and Recovery
It evaluates the tendency to suffer muscle and tendon injuries, as well as the ability to recover. Key genes: ACTN3, COL1A1, COL5A1, EMILIN1, GDF5, IL6, MNSOD, TNFa.

Muscular Hypertrophy
It meases the genetic capacity for muscle growth and the response to strength training. Key genes: ACTN3, PPARa, PPARg, VEGFa, MTHFR, GDF5.

Oxidative Stress Tolerance and Recovery Evaluates the body's ability to neutralize damage caused by free radicals during intense exercise and efficiency in muscle recovery: SOD2, CAT, NRF2

Inflammation: Immune Response: Evaluates the genetic predisposition to inflammation and the immune response after exercise, key factors in the recovery and prevention of injuries: CRP, TNF, IL1B

Oxygenation Capacity (VO2max)
It indicates the efficiency of transport and use of oxygen during exercise, key in endurance sports. Key genes: ACE, ACTN3, HIF1A, VEGFa, NRF2, PPARa

Weight Regulation and nutrition

The choice of an adequate diet for body weight control adapted to energy consumption is essential to achieve sports objectives in an effective and safe way, since we do not all respond in the same way to the same type of diet and food.

It analyzes the predisposition to metabolize fats and carbohydrates, and the body's response to exercise for weight loss.

Key genes: ADRB2, APOA2, APOA5,   CRP, FTO, GHRL, GNB3, MC4R, NPY, PPARa, PPARg, TRHR, UCP2.

Nutrigenetic test:

MyDiet is a genetic test that allows you to determine the nutrigenetic profile of people to improve the personalization of dietary-sports plans and increase their effectiveness.

It helps in weight loss by identifying how your genetics influence the metabolism of fats and carbohydrates, the response to exercise, satiety and the predisposition to obesity. With this information, dietary and training strategies can be customized to optimize fat burning and improve adherence to an effective nutritional plan.

Obesity usually has a multifactorial origin, combining genetic factors and the modern lifestyle. Each person responds differently to the same diet and exercise, which explains why some maintain their weight effortlessly, while others struggle to lose weight. Although habits are key, genetics also influence weight control and body composition.

What is nutrigenetics?

Nutrigenetics is a discipline of nutritional genomics that studies how variations in DNA influence the way each person metabolizes nutrients and responds to food. These genetic differences, which are not always evident, may explain why two people who follow a similar diet may have very different levels of cholesterol, blood pressure or body composition.

Due to these variations in our genetic makeup, each individual has unique nutritional needs that cannot be generalized. This highlights the importance of designing personalized nutritional plans, based not only on visible symptoms, but also on genetic information that helps us better understand how to optimize health and well-being.

Regulation of Carbohydrate Metabolism, Key Genes:

ACE : Predisposition to greater cardiovascular efficiency.

GLUT2: Related to greater glucose absorption and predisposition to blood sugar peaks. It can increase the risk of insulin resistance if the diet is not well managed.

PLIN1-3: It regulates carbohydrate metabolism and insulin response. It can influence fat storage in response to carbohydrates.

TAS1R2:Related to sweet taste perception, which can influence the preference for refined carbohydrates.

TCF7L2: Association with lower risk of type 2 diabetes and better response to insulin. Relationship between fat and sugar metabolism.

MyDiet is a genetic test that analyzes genetic markers of the main genes involved in:

Regulation of Appetite and Satiety: Key genes:

NPY:Associated with lower production of Neuropeptide Y, which can reduce the tendency to overfeed and excessive caloric intake,

GHRL: Related to low levels of ghrelin, which may mean a lower hunger boost.

MC4R: It can influence the regulation of carbohydrate metabolism and increased insulin sensitivity. Regulation of appetite, feeling of satiety

DRD2 : Variant related to a greater tendency to gluttony and reward for foods rich in fat and carbohydrates.

Sleep regulation

Propensity to addictions and gluttony

Impact of the exercise: Key genes:

Risk of Obesity: Key genes:

FTO (1, 2, 3):Associated with the genetic risk of obesity and overweight. It influences the ability to satiety, appetite and the predisposition to accumulate body fat, especially in response to high-calorie diets.

APOA2 / APOA5: Genes related to fat metabolism and the response to the intake of saturated fats and cholesterol. They can condition the tendency to have high levels of triglycerides or cholesterol depending on the type of diet.

FABP2: Regulates the absorption of fatty acids in the intestine. Variants in this gene can increase the uptake of dietary fats, favoring their storage and increasing metabolic risk.

PPARA / PPARG: Regulation of fat oxidation and energy storage. They determine the efficiency to burn fat and metabolize lipids, influencing the response to diets and the predisposition to accumulate adipose tissue.

ADRB2 / ADRB3: Key adrenergic receptors in lipolysis and thermogenesis. They regulate the ability to release and burn fat, especially in response to physical exercise and the activation of the sympathetic nervous system.

PLIN1 (1, 2, 3): They control the release of fat stored in adipocytes. Variants in these genes can hinder the mobilization of body fat, affecting the effectiveness of weight loss with diet and exercise.

PPGC1A: Ability to generate heat and burn fat (thermogenesis).

DRD2: Associated with lower dopaminergic sensitivity, greater risk of addictions and impulsive eating behaviors.

NPY: Linked to greater emotional appetite and tendency to overeat under stress.

FTO: Related to less feeling of satiety and greater caloric intake, especially fats.

TAS1R2: Predisposes to a greater preference for sweet foods and simple carbohydrates.

GHRL:It modulates the feeling of hunger through the hormone ghrelin.

MC4R: Regulates satiety; mutations can favor hyperphagia.

CLOCK: it is involved in the circadian rhythm, regulates the sleep and wakefulness cycles, and its alteration is associated with a greater risk of obesity, imbalances in satiety and disregulation of appetite, especially in people with nocturnal habits or shift work.

IMPROVES THE EFFECTIVENESS OF SUPPLEMENTS AND VITAMINS THROUGH GENETICS:

The MySupplements test is a genetic test that analyzes genetic markers involved in supplements and vitamins:

• Increase available energy levels

• Regulate blood cholesterol and triglyceride levels

  Control the feeling of hunger and appetite

• Help prevent the accumulation of oxidizing substances in the body

• Facilitate the metabolism of fats

• Prevent the inflammatory response

• Prevent the appearance of injuries

• Promote the acquisition and quality of sleep

• Regulate levels of vitamin B9 (folate) and B12

• Improve vasodilation

The MySupplements test analyzes the following supplements and vitamins:

Vitamins:

1. Vitamin B1 (Tiamine)

2. Vitamin B2 (Riboflavin)

3. Vitamin B3 (Niacin)

4. Vitamin B9 (Folate)

5. Vitamin B12

6. Vitamin C

7. Vitamin E

8. Conjugated Linoleic Acid (CLA)

9. Ursolic acid

10. Amylopectin

11. BCAA

12. Caffeine

13. Catechin

14. Chitosan

15. Coenzyme Q10

16. Chondroitin

17. Curcumin

18. Spirulina

19. Fucoxanthin

20. Garcinia Cambogia

21. Genistein

22. Glucomanan

23. Glucose

24. Glucosamine

25. Glutamine

26. HMB

27. Irvingia gabonensis

28. L-Arginine

29. L-Carnitine

30. L-Citrulline

31. Lysine

32. L-Tyrosine

33. L-Tryptophan

34. Magnesium

35. Maltodextrin

36. Melatonin

37. Methylsulfonylmethane (MSM)

38. Creatine monohydrate

39. Organic nitrates

40. Omega 3

41. Chromium Picolinate

42. Proline

43. Proteins

44. Quercetin

45. Zinc

Hormonal health test:

• MyEstrogen is a genetic test that determines the activity of enzymes related to estrogen metabolism

• MyEstrogen helps to better understand hormonal health, prevent and manage conditions related to an imbalance in estrogen levels.

Who is My Estrogen for?

To all professionals interested in the prevention of diseases and conditions related to a hormonal imbalance in both men and women, especially those related to estrogen levels: cancer (estrogen, breast, prostate ...), polycystic ovary, endometriosis, irregular menstruation, premenstrual syndrome, aromatase deficiency, alopecia, prostatic hypertophy, etc.

What do analyze?

The test analyzes genetic markers involved in estrogen metabolism: CYP17A1, CYP19A1, CYP1A1, CYP1B1, CYP3A4, COMT, NQO1, SULT1A1, GSTM1, GSTT1, SOD2.

My Detox

Phase II – Conjugation and neutralization of toxins

It converts active metabolites into soluble and non-toxic substances that can be eliminated.

COMT → Methylation: Detoxifies catecholamines and estrogens. Polymorphisms can induce hyperreactivity to stress, chronic pain and neurovegetative dysfunction.

TPMT → Methylation of thopurines

GSTM1, GSTM3, GSTT1, GSTP1 →Conjugation with glutathione: they also eliminate heavy metals, xenobiotics and free radicals. Neutralizes for g: mercury and aluminum.

NAT2 → Acetilation

SULT1A1 → Sulfation

UGT1A1 → Glucuronidation

🔍 ¿What is analyzed?

• Ability to neutralize free radicals

• Detoxification of drugs, heavy metals, pesticides, histamine, amines, alcohol

• Elimination of hormones such as estrogen and derivatives of dopamine metabolism and catecholamines

Phase I - Activation of toxins (oxidation, hydrolysis, reduction)

These genes transform lipophilic toxins into reactive intermediates.

• CYP1A2

• CYP2C9

• CYP2C19

• CYP2D6

• CYP3A4

• CYP3A5:

🔍 ¿What is analyzed?

• Metabolism of drugs, caffeine, alcohol and xenobiotics

• Conversion of estrogens into catechelestrogens

• Variability in the rate of metabolism (slow, intermediate or ultra-fast metabolizer)

Estrogen metabolism

The test includes genes that regulate the transformation, neutralization and elimination of estrogens and their derivatives

• CYP1A2, CYP3A4, CYP3A5 → Estrogen hydroxylation (phase I)

• COMT → Methylation of catechoestrogens

• SULT1A1 y UGT1A1 → Sulfation and glucuronidation for excretion

• GSTs → Detoxification of prooxidant metabolites

🔍 ¿What is analyzed?

• Tendency to build up active estrogens

• Risk of estrogenic dominance or sensitivity to hormone therapies

• Ability to eliminate pro-inflammatory metabolites derived from estrogen metabolism

Metabolism of drugs

Many of the enzymes studied participate in the activation, deactivation or elimination of drugs.

CYPs (1A2, 2C9, 2C19, 2D6, 3A4, 3A5) → Main drug metabolization system in phase 2

TPMT, NAT2, UGT1A1, SULT1A1, GSTs →Phase II conjugation processes

🔍 ¿What is analyzed?

• Possible adverse reactions or toxicity to common drugs

• Speed of drug elimination (prolonged or insufficient effects)

• Application in pharmacogenetics for personalized medicine

Detoxification of free radicals and environmental toxins

Evaluates antioxidant capacity and response to environmental pollutants, heavy metals and oxidative stress.

GSTM1, GSTM3, GSTT1, GSTP1

COMT (Indirect antioxidant)

NAT2, UGT1A1, SULT1A1

🔍 ¿What is analyzed?

• Ability to neutralize free radicals

• Removal of xenobiotics, environmental toxins, pollution, additives, solvents

• Risk of toxic accumulation and cell inflammation

MyDetox offers a key tool to anticipate, prevent and design truly personalized liver health protocols.

This test is useful in people who have:

• Chronic fatigue or persistent mental fog

• Neurocognitive and autism spectrum disorders

• Intolerance to medications, supplements or anesthesia

• Adverse reactions to common drugs (such as paracetamol, NSAIDs, anxiolytics)

• Recurrent digestive problems with no apparent cause

• Multiple chemical sensitivity (perfumes, pollutants, pesticides)

• Treatment-resistant headaches, migraines or acne outbreaks

• Histaminosis, alcohol intolerance or difficulty tolerating certain meals

• Hormonal disorders (estrogen dominance, severe premenstrual syndrome, symptomatic menopause)

• Chronic inflammatory or autoimmune diseases

• Family history of hormone-dependent cancer or liver disease

🔍In these cases, it is possible that there are genetic polymorphisms that affect liver function, limiting the body's ability to eliminate toxins efficiently.

My Neuro

Optimizes mental health, understands emotional balance, stress response and brain function by analyzing key genetic variants.